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A MORBIDLY OBESE PATIENT WITH MEAT STUCK IN HIS ESOPHAGUS

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You’re the anesthesiologist on call on a Saturday night. A patient arrives at the Emergency Room complaining that he ate piece of steak one hour ago, and the meat got stuck in his throat. He is morbidly obese patient who stands six feet tall and weighs 350 pounds, for a BMI of 47.

The attending general surgeon wants to do an upper GI endoscopy to extract the piece of meat from the patient’s esophagus or push it through into the stomach. He’s called on you to do the anesthetic. 

What do you do?

You examine the patient and find he has a short neck, a small mouth, and a large tongue. You cannot see his soft palate at all, and you rate him as a Mallampati 4.

Mallampati Class IV airway

The patient is alert, and is an excellent historian. He cannot even swallow his saliva. He has no difficulty breathing or significant chest pain. His hospital chart shows no past anesthetics, and he has no medical problems except hypertension which is treated with lisinopril. His vital signs are normal, and his oxygen saturation is 96% on room air.You are six months out of anesthesia training and new to this hospital. The surgeon—a 60-year-old male with the brash confidence of General Patton—is an iconic and respected figure at this medical center. He wants to proceed at once. It’s 8 pm on a Saturday night. He requests “just a little sedation” so he can insert the endoscope past the gag reflex and into the esophagus.

You bring the patient into the endoscopy suite, attach the standard vital signs monitors, and administer oxygen via a Procedural Oxygen Mask (POM, made by Mercury Medical).

You administer 2 mg of Versed and 100 micrograms of fentanyl IV. The surgeon sprays Cetacaine into the patient’s mouth for topical anesthesia and inserts a bite block. After five minutes time the patient is still wide awake. The surgeon looks at you and says, “I need him a little deeper than this.” You administer another 1 mg of Versed and 50 micrograms of fentanyl. After another five minutes time, the patient is still wide awake. The surgeon looks at you and repeats, “I need him a little deeper than this.” He says this in an impatient condescending tone, and you feel pressured. You administer 50 mg of propofol, and the patient’s eyes begin to drift closed. The surgeon inserts the gastroscope, after which the patient coughs, gags, and vomits into his airway. His oxygen saturation which had been 100% quickly plummets to 75%. You move to the head of the bed, suction the patient’s mouth, and attempt bag-mask ventilation without success. His oxygen saturation drops to 60%. You reach for a Miller 3 laryngoscope and attempt to intubate the trachea, but you cannot visualize his vocal cords. You are panicked. The surgeon is screaming at you to do something. You tell the surgeon he needs to do a tracheostomy. In the meantime you insert a laryngeal mask airway into the patient’s throat, but are still unable to ventilate the lungs. The ECG rhythm converts to ventricular fibrillation, and you call a Code Blue.

After thirty minutes of CPR and ACLS, the patient is declared dead.

What went wrong here? A patient who walked into the hospital is now dead. The basic problem was that the anesthesiologist proceeded to deeply sedate a patient with a full stomach (a known aspiration risk) without first controlling the airway by inserting an endotracheal tube. This morbidly obese patient with a thick neck, a small mouth, and a large tongue was always going to be difficult to intubate, but a successful intubation was most likely to occur under controlled circumstances with the patient awake prior to any endoscopy. The issue of a domineering surgeon pushing an inexperienced anesthesia provider into doing the wrong anesthetic is a key problem. This can and does happen, and once the case has concluded with a bad outcome, that same surgeon will deny any culpability, step back and say “I don’t do anesthesia. The decisions and actions of the anesthesiologist caused the problem, not me.”

How should the anesthetic have been done? 

In a parallel universe, an experienced anesthesiologist would do the following:

What are the lessons to be learned from this case study?

You may need it to save someone’s life one day.

Footnote:

A. (From Chapter 44, Airway Management in Adults, Miller’s Anesthesia, Ninth edition, pp 1373-1412)  “Topical application of local anesthetic to the airway should, in most cases, be the primary anesthetic for awake airway management. Lidocaine is the most commonly used local anesthetic for awake airway management because of its rapid onset, high therapeutic index, and availability in a wide variety of preparations and concentrations. Benzocaine and Cetacaine (a topical application spray containing benzocaine, tetracaine, and butamben; Cetylite Industries, Pennsauken, NJ) provide excellent topical anesthesia of the airway, but their use is limited by the risk of methemoglobinemia, which can occur with as little as 1 to 2 seconds of spraying. . . .  A mixture of lidocaine 3% and phenylephrine 0.25%, which can be made by combining lidocaine 4% and phenylephrine 1% in a 3:1 ratio, has similar anesthetic and vasoconstrictive properties as topical cocaine and can be used as a substitute. Topical application of local anesthetic should primarily be focused on the base of the tongue (pressure receptors here act as the afferent component of the gag reflex), the oropharynx, the hypopharynx, and the laryngeal structures; anesthesia of the oral cavity is unnecessary. . . . Before topical application of local anesthetic to the airway, administration of an anticholinergic agent should be considered to aid in the drying of secretions, which helps improve both the effectiveness of the topical local anesthetic and visualization during laryngoscopy. Glycopyrrolate is usually preferred because it has less vagolytic effects than atropine at doses that inhibit secretions and does not cross the blood-brain barrier. It should be administered as early as possible to maximize its effectiveness. “. . . Oropharyngeal anesthesia can be achieved by the direct application of local anesthetic or by the use of an atomizer or nebulizer. Topical application of local anesthetic to the larynx can be achieved by directed atomization of a local anesthetic or by the  spray-as-you-go (SAYGO) method, which involves intermittently injecting local anesthetic through the suction port or working channel of a flexible intubation scope (FIS) or optical stylet, as it is advanced toward the trachea.“Topical application of local anesthetic to the airway mucosa using one or more of these methods is often sufficient. If supplemental anesthesia is required, then a variety of nerve blocks may be used. Three of the most useful are the glossopharyngeal nerve block, superior laryngeal nerve block, and translaryngeal block. The glossopharyngeal nerve supplies sensory innervation to the posterior third of the tongue, vallecula, the anterior surface of the epiglottis, and the posterior and lateral walls of the pharynx, and is the afferent pathway of the gag reflex. To block this nerve, the tongue is displaced medially, forming a gutter (glossogingival groove). A 25-gauge spinal needle is inserted at the base of the anterior tonsillar pillar, just lateral to the base of the tongue, to a depth of 0.5 cm. After negative aspiration for blood or air, 2 mL of 2% lidocaine is injected. The process is then repeated on the contralateral side. The same procedure can be performed noninvasively with cotton-tipped swabs soaked in 4% lidocaine; the swabs are held in place for 5 minutes.”     

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